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Becilan (Pyridostigmine)

Becilan belongs to a group of medicines called cholinergic antimyasthenics. It works by preventing the breakdown of a natural chemical substance in your body called acetylcholine. This chemical is needed to stimulate muscle movement.

Other names for this medication:
Pyridostigmine, Mestinon, Becilan, Distinon, Piridostigmina, Regonol,

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Becilan comes as a regular tablet, an extended-release (long-acting) tablet, and a syrup to take by mouth. It usually is taken once, twice, or several times a day, depending on the type of tablet. Your doctor may change your dose, depending on how you respond to the drug. When you first start taking Becilan, your doctor may want you to keep a daily record of the time you take each dose, how long you feel better after taking each dose, and if you have side effects. This record will help the doctor decide how much drug is best for you. Follow the directions on your prescription label carefully, and ask your doctor or pharmacist to explain any part you do not understand. Take Becilan exactly as directed. Do not take more or less of it or take it more often than prescribed by your doctor.

Swallow the extended-release tablets whole; do not chew or crush them.

Continue to take Becilan even if you feel well. Do not stop taking Becilan without talking to your doctor.

Becilan overdose can cause severe illness, including muscle weakness. It is very hard to tell the difference between too little and too much Becilan. Call your doctor immediately if your symptoms become worse.

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STUDY OF THE NUCLEIC ACIDS IN THE BRAIN, CEREBELLUM AND LIVER AS A FUNCTION OF THE AGE OF THE ANIMAL AND UNDER THE INFLUENCE OF TREATMENT WITH VITAMIN B 12, VITAMIN B 6 AND FOLIC thuoc zosert 100mg to ml ACID Social functioning, quality of life, and psychopathological evaluation were performed with validated tools. The antibiotic sensitivity of 4095 Salmonella typhi strains isolated in Hungary from January, 1974 to June 1979, was tested. A simple radioimmunoassay to determine binding of antibodies to lipid antigens. Here we show that a p43 depletion in mice decreases mitochondrial DNA replication and respiratory chain activity in skeletal muscle in association with the induction of a more glycolytic muscle phenotype and a decrease of capillary density.

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